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Article 2016-04

Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas

Autor: Morales, M.G. Abrigo, J. Acuna, M.J. Santos, R.A. Bader, M. Brandan, E. Simon, F. Olguin, H. Cabrera, D. Cabello-Verrugio, C.

Immobilization is a form of disuse characterized by a loss of strength and muscle mass. Among the main features are decreased IGF-1/Akt signalling and increased ubiquitin-proteasome pathway signalling, which induce greater myosin heavy chain degradation. Activation of the classical renin-angioten...

Enlace original: http://repositorio.unab.cl/xmlui/handle/ria/3994

Repositorio Universidad Andrés Bello

Angiotensin-(1-7)

Atrophy

Disuse

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Morales, M.G. , Abrigo, J. , Acuna, M.J. , Santos, R.A. , Bader, M. , Brandan, E. , Simon, F. , Olguin, H. , Cabrera, D. , Cabello-Verrugio, C. , [Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas] , Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas

Artículo 2014

Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis

Autor: Cabrera, D. Gutiérrez, J. Cabello-Verrugio, C. Morales, M. G. Mezzano, S. Fadic, R. Casar, J. C. Hancke, J. L. Brandan, E.

Background: Duchenne muscular dystrophy (DMD) is characterized by the absence of the cytoskeletal protein dystrophin, muscle wasting, increased transforming growth factor type beta (TGF-β) signaling, and fibrosis. At the present time, the only clinically validated treatments for DMD are glucocort...

Enlace original: https://repositorio.uc.cl/handle/11534/27461 https://repositorio.uc.cl/handle/11534/27461

Repositorio UC

Andrographolide

Cell therapy

DMD

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Cabrera, D. , Gutiérrez, J. , Cabello-Verrugio, C. , Morales, M. G. , Mezzano, S. , Fadic, R. , Casar, J. C. , Hancke, J. L. , Brandan, E. , [Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis] , Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis

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Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas

Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis

Descubre y comparte nuestro conocimiento público

Resultados de búsqueda

Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas

[Morales_M_Angiotensin.pdf]

Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis

[Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis.pdf]