Autor:
Vecchiola Cárdenas, Andrea Paola.
Lagos Arévalo, Carlos Fernando.
Fuentes, Cristóbal A.
Allende, Fidel.
Campino Johnson, María del Carmen.
Valdivia, Carolina.
Tapia Castillo, Alejandra.
Owen, Gareth Ivor.
Solari Gajardo, Sandra.
Carvajal Maldonado, Cristián Andrés.
Fardella B., Carlos.
Ogishima, Tadashi.
Mukai, Kuniaki.
Abstract Background Familial hyperaldosteronism type I (FH-I) is caused by the unequal recombination between the 11beta-hydroxylase (CYP11B1) and aldosterone synthase (CYP11B2) genes, resulting in the generation of a CYP11B1/B2 chimeric gene and abno...
Enlace original:
https://repositorio.uc.cl/handle/11534/26778
Vecchiola Cárdenas, Andrea Paola.
,
Lagos Arévalo, Carlos Fernando.
,
Fuentes, Cristóbal A.
,
Allende, Fidel.
,
Campino Johnson, María del Carmen.
,
Valdivia, Carolina.
,
Tapia Castillo, Alejandra.
,
Owen, Gareth Ivor.
,
Solari Gajardo, Sandra.
,
Carvajal Maldonado, Cristián Andrés.
,
Fardella B., Carlos.
,
Ogishima, Tadashi.
,
Mukai, Kuniaki.
,
[Different effects of progesterone and estradiol on chimeric and wild type aldosterone synthase in vitro]
,
Different effects of progesterone and estradiol on chimeric and wild type aldosterone synthase in vitro
